By Hermann Steller
Apoptosis and Development, the most recent quantity of Current subject matters in Developmental Biology keeps the legacy of this prime serial with caliber chapters authored via leaders within the box.
This quantity covers learn tools in apoptosis and improvement, and comprises sections on such issues because the non-lethal function of apoptotic proteins and germ line mobile demise in Drosophila.
- Continues the legacy of this ultimate serial with caliber chapters authored by way of leaders within the field
- Includes descriptions of the latest advances within the field
- Covers examine equipment in apoptosis and improvement, and comprises sections on such subject matters because the non-lethal function of apoptotic proteins and germ line mobilephone loss of life in Drosophila
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Chalfie, M. (2007). Targeted cell killing by reconstituted caspases. Proceedings of the National Academy of Sciences of the United States of America, 104, 2283–2288. , Hersh, B. , et al. (2000). Translocation of C. elegans CED-4 to nuclear membranes during programmed cell death. Science, 287, 1485–1489. Chen, Y. , Lee, E. , Skeen-Gaar, R. , & Xue, D. (2013). Caspase-mediated activation of Caenorhabditis elegans CED-8 promotes apoptosis and phosphatidylserine externalization. Nature Communications, 4, 2726.
Egl-1(lf ) and ced-9(gf ) mutations block germ cell death in response to ionizing radiation (Gartner, Milstein, Ahmed, Hodgkin, & Hengartner, 2000); however, as in the tail-spike cell, neither mutation affects normal physiological cell death in the germ line. Other regulators appear to take charge here (Fig. 2). Mutants in pax-2 and egl-38, encoding Pax2/5/8 family transcriptional regulators, have increased rates of germ cell death. Overexpression of these genes reduces germline apoptosis (Park, Jia, Rajakumar, & Chamberlin, 2006).
Elegans CED-9 in the absence of the C-terminal transmembrane domain. Cell Death and Differentiation, 14, 1925–1935. Tan, F. , Zuckerman, J. , Wells, R. , & Hill, R. B. (2011). The C. elegans B-cell lymphoma 2 (Bcl-2) homolog cell death abnormal 9 (CED-9) associates with and remodels LIPID membranes. Protein Science, 20, 62–74. , & Conradt, B. (2003). The Snail-like CES-1 protein of C. elegans can block the expression of the BH3-only cell-death activator gene egl-1 by antagonizing the function of bHLH proteins.